Omi / HtrA2 is relevant to the selective vulnerability of striatal neurons in Huntington’s disease-Reference-Cited by-同舟云学术

Omi / HtrA2 is relevant to the selective vulnerability of striatal neurons in Huntington’s disease

Published:2008-07 Issue:1 Volume:28 Page:30-40
ISSN:0953-816X
Container-title:European Journal of Neuroscience
language:en
Short-container-title:

Author:

Inagaki Reina,Tagawa Kazuhiko,Qi Mei-Ling,Enokido Yasushi,Ito Hikaru,Tamura Takuya,Shimizu Shigeomi,Oyanagi Kityomitsu,Arai Nobutaka,Kanazawa Ichiro,Wanker Erich E,Okazawa Hitoshi

Publisher

Wiley

Subject

General Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1460-9568.2008.06323.x/fullpdf

Reference32 articles.

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2. Hax1-mediated processing of HtrA2 by Parl allows survival of lymphocytes and neurons;Chao;Nature,2008

3. The HtrA family of proteases: implication for protein composition and cell fate;Clausen;Mol. Cell,2002

4. Akt is altered in an animal model of Huntington’s disease and in patients;Colin;Eur. J. Neurosci.,2005

5. The C-terminal tail of presenilin regulates Omi/HtrA2 protease activity;Gupta;J. Biol. Chem.,2004

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