Increased intrarenal post‐glomerular blood flow is a key condition for the development of calcineurin inhibitor‐induced renal tubular acidosis in kidney transplant recipients-Reference-Cited by-同舟云学术

Increased intrarenal post‐glomerular blood flow is a key condition for the development of calcineurin inhibitor‐induced renal tubular acidosis in kidney transplant recipients

Published:2022-03-20 Issue:6 Volume:36 Page:
ISSN:0902-0063
Container-title:Clinical Transplantation
language:en
Short-container-title:Clinical Transplantation

Author:

Kaneko Shuzo¹²^ORCID,Usui Joichi¹,Takahashi Kazuhiro³^ORCID,Oda Tatsuya³,Yamagata Kunihiro¹

Affiliation:

1. Department of Nephrology Faculty of Medicine University of Tsukuba Ibaraki Japan

2. Department of Nephrology Itabashi Chuo Medical Center Itabashi Tokyo Japan

3. Department of Gastroenterological and Hepatobiliary Surgery and Organ Transplantation University of Tsukuba Tsukuba Ibaraki Japan

Publisher

Wiley

Subject

Transplantation

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/ctr.14648

Reference44 articles.

1. Renal tubular acidosis after kidney transplantation--incidence, risk factors and clinical implications

2. Prevalence and risk factors of renal tubular acidosis after kidney transplantation;Malik SI;J Pak Med Assoc,2011

3. Metabolic acidosis in renal transplantation: neglected but of potential clinical relevance

4. Posttransplant metabolic acidosis: a neglected factor in renal transplantation?

5. Hyperchloremic metabolic acidosis in the kidney transplant patient

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1. Metabolic Acidosis Is Associated With an Accelerated Decline of Allograft Function in Pediatric Kidney Transplantation;Kidney International Reports;2024-06

2. Metabolic acidosis post kidney transplantation;Frontiers in Physiology;2022-08-23