Soluble analog of ApoER2 targeting beta2-glycoprotein I in immune complexes counteracts hypertension in lupus-prone mice with spontaneous antiphospholipid syndrome
Author:
Affiliation:
1. Department of Medicine; Beth Israel Deaconess Medical Center and Harvard Medical School; Boston MA USA
Funder
National Institutes of Health
Lupus Research Institute
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/jth.13314/fullpdf
Reference65 articles.
1. International consensus statement on an update of the classification criteria for definite antiphospholipid syndrome (APS);Miyakis;J Thromb Haemost,2006
2. 14th International Congress on Antiphospholipid Antibodies: task force report on antiphospholipid syndrome treatment trends;Erkan;Autoimmun Rev,2014
3. Therapy: antiphospholipid syndrome research needs more collaboration;Erkan;Nat Rev Rheumatol,2014
4. Thrombotic recurrences and bleeding events in APS vascular patients: a review from the literature and a comparison with the APS Piedmont Cohort;Bazzan;Autoimmun Rev,2013
5. Morbidity and mortality in the antiphospholipid syndrome during a 10-year period: a multicentre prospective study of 1000 patients;Cervera;Ann Rheum Dis,2015
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1. Protection by hydroxychloroquine prevents placental injury in obstetric antiphospholipid syndrome;Journal of Cellular and Molecular Medicine;2022-06-29
2. PEGylated Domain I of Beta-2-Glycoprotein I Inhibits Thrombosis in a Chronic Mouse Model of the Antiphospholipid Syndrome;Frontiers in Immunology;2022-04-11
3. 16th International Congress on Antiphospholipid Antibodies Task Force Report on Antiphospholipid Syndrome Treatment Trends;Lupus;2020-10
4. The role of beta-2-glycoprotein I in health and disease associating structure with function: More than just APS;Blood Reviews;2020-01
5. Domain I of β2GPI is capable of blocking serum IgA antiphospholipid antibodies binding in vitro: an effect enhanced by PEGylation;Lupus;2019-05-24
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