NLRP6‐caspase 4 inflammasome activation in response to cariogenic bacterial lipoteichoic acid in human dental pulp inflammation-Reference-Cited by-同舟云学术

NLRP6‐caspase 4 inflammasome activation in response to cariogenic bacterial lipoteichoic acid in human dental pulp inflammation

Published:2021-01-20 Issue:6 Volume:54 Page:916-925
ISSN:0143-2885
Container-title:International Endodontic Journal
language:en
Short-container-title:Int Endod J

Author:

Tian X. X.¹^ORCID,Li R.¹,Liu C.²,Liu F.¹,Yang L. J.¹,Wang S. P.¹,Wang C. L.³

Affiliation:

1. Stomatology Center The First Affiliated Hospital of Zhengzhou University ZhengzhouChina

2. Department of Interventional Neuroradiology The First Affiliated Hospital of Zhengzhou University ZhengzhouChina

3. State Key Laboratory of Oral Diseases National Clinical Research Center for Oral Diseases Department of Cariology and Endodontics West China Hospital of Stomatology Sichuan University Chengdu China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

General Dentistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/iej.13469

Reference31 articles.

1. NALP3 Forms an IL-1β-Processing Inflammasome with Increased Activity in Muckle-Wells Autoinflammatory Disorder

2. Dysregulation of Inflammasomes in Human Dental Pulp Cells Exposed to Porphyromonas gingivalis and Fusobacterium nucleatum

3. MCC950 directly targets the NLRP3 ATP-hydrolysis motif for inflammasome inhibition

4. A small-molecule inhibitor of the NLRP3 inflammasome for the treatment of inflammatory diseases

5. Platelet-activating factor (PAF) mediates NLRP3-NEK7 inflammasome induction independently of PAFR

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