Amyloid‐β oligomers in cellular models of Alzheimer’s disease-Reference-Cited by-同舟云学术

Amyloid‐β oligomers in cellular models of Alzheimer’s disease

Published:2020-05-18 Issue:4 Volume:155 Page:348-369
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Fontana Igor C.¹²^ORCID,Zimmer Aline R.¹^ORCID,Rocha Andreia S.²^ORCID,Gosmann Grace¹^ORCID,Souza Diogo O.²³^ORCID,Lourenco Mychael V.⁴^ORCID,Ferreira Sergio T.⁴⁵^ORCID,Zimmer Eduardo R.²⁶⁷^ORCID

Affiliation:

1. Faculty of Pharmacy Universidade Federal do Rio Grande do Sul (UFRGS) Porto Alegre Brazil

2. Graduate Program in Biological Sciences: Biochemistry UFRGS Porto Alegre Brazil

3. Department of Biochemistry UFRGS Porto Alegre Brazil

4. Institute of Medical Biochemistry Leopoldo de Meis Federal University of Rio de Janeiro Rio de Janeiro Brazil

5. Institute of Biophysics Carlos Chagas Filho Federal University of Rio de Janeiro Rio de Janeiro Brazil

6. Department of Pharmacology UFRGS Porto Alegre Brazil

7. Graduate Program in Biological Sciences: Pharmacology and Therapeutics, UFRGS Porto Alegre Brazil

Funder

Fundação Carlos Chagas Filho de Amparo à Pesquisa do Estado do Rio de Janeiro

Fundação de Amparo à Pesquisa do Estado do Rio Grande do Sul

Alzheimer's Association

Conselho Nacional de Desenvolvimento Científico e Tecnológico

Coordenação de Aperfeiçoamento de Pessoal de Nível Superior

International Society for Neurochemistry

Institute for Translational Neuroscience

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jnc.15030

Reference223 articles.

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2. -Amyloid Peptides Induce Mitochondrial Dysfunction and Oxidative Stress in Astrocytes and Death of Neurons through Activation of NADPH Oxidase

3. Amyloid β attenuates metabotropic zinc sensing receptor, mZnR/GPR39, dependent Ca2+ , ERK1/2 and Clusterin signaling in neurons

4. An In Vitro Model for Neuroscience: Differentiation of SH-SY5Y Cells into Cells with Morphological and Biochemical Characteristics of Mature Neurons

5. Structural conversion of neurotoxic amyloid-β1–42 oligomers to fibrils

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