Progression in multiple sclerosis is associated with low endogenous NCAM-Reference-Cited by-同舟云学术

Progression in multiple sclerosis is associated with low endogenous NCAM

Published:2013-04-19 Issue:5 Volume:125 Page:766-773
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Gnanapavan Sharmilee¹²,Ho Peggy³,Heywood Wendy⁴,Jackson Sam²,Grant Donna¹,Rantell Khadija⁵,Keir Geoff¹,Mills Kevin⁴,Steinman Lawrence³,Giovannoni Gavin²

Affiliation:

1. Department of Neuroimmunology; Institute of Neurology; London UK

2. Blizard Institute of Cell and Molecular Science; Barts and The London School of Medicine and Dentistry; Queen Mary University of London; London UK

3. Department of Neurology and Neurological Sciences; Stanford University School of Medicine; Stanford California USA

4. Biochemistry Group; Clinical and Molecular Genetics Unit; Institute of Child Health; London UK

5. Statistician; Institute of Neurology; London UK

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.12236/fullpdf

Reference33 articles.

1. Characterization of soluble forms of NCAM;Bock;FEBS Lett.,1987

2. Age at disability milestones in multiple sclerosis;Confavreux;Brain,2006

3. Developmental regulation of neural cell adhesion molecule in human prefrontal cortex;Cox;Neuroscience,2009

4. Inactivation of the N-CAM gene in mice results in size reduction of the olfactory bulb and deficits in spatial learning;Cremer;Nature,1994

5. NCAM is essential for axonal growth and fasciculation in the hippocampus;Cremer;Mol. Cell. Neurosci.,1997

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