Comparative idiosyncrasies in life extension by reduced mTOR signalling and its distinctiveness from dietary restriction-Reference-Cited by-同舟云学术

Comparative idiosyncrasies in life extension by reduced mTOR signalling and its distinctiveness from dietary restriction

Published:2016-05-03 Issue:4 Volume:15 Page:737-743
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Garratt Michael¹,Nakagawa Shinichi²³^ORCID,Simons Mirre J. P.⁴

Affiliation:

1. Department of Pathology; University of Michigan Medical School; Ann Arbor MI 48109 USA

2. Evolution and Ecology Research Group and School of Biological; Earth and Environmental Sciences; The University of New South Wales; Sydney NSW 2052 Australia

3. Diabetes and Metabolism Division; Garvan Institute of Medical Research; Sydney NSW 2010 Australia

4. Department of Animal and Plant Sciences; University of Sheffield; Sheffield S10 2TN UK

Funder

Michigan Society of Fellows

ARC Future Fellowship (Australia)

Natural Environment Research Council

Sir Henry Wellcome and Sheffield Vice Chancellor's fellowships

Publisher

Wiley

Subject

Cell Biology,Aging

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/acel.12489/fullpdf

Reference46 articles.

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2. Mechanisms of life span extension by rapamycin in the fruit fly Drosophila melanogaster;Bjedov;Cell Metab.,2010

3. drr-2 encodes an eIF4H that acts downstream of TOR in diet-restriction-induced longevity of C. elegans;Ching;Aging Cell,2010

4. Assessment of Mitochondrial Biogenesis and mTORC1 Signaling During Chronic Rapamycin Feeding in Male and Female Mice;Drake;J. Gerontol. A Biol. Sci. Med. Sci.,2013

5. Target of rapamycin signalling mediates the lifespan-extending effects of dietary restriction by essential amino acid alteration;Emran;Aging (Albany NY),2014

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