GSTO1‐1 is an upstream suppressor of M2 macrophage skewing and HIF‐1α‐induced eosinophilic airway inflammation-Reference-Cited by-同舟云学术

GSTO1‐1 is an upstream suppressor of M2 macrophage skewing and HIF‐1α‐induced eosinophilic airway inflammation

Published:2020-03-02 Issue:5 Volume:50 Page:609-624
ISSN:0954-7894
Container-title:Clinical & Experimental Allergy
language:en
Short-container-title:Clin Exp Allergy

Author:

Sokulsky Leon A.¹²^ORCID,Goggins Bridie¹²,Sherwin Simonne¹²,Eyers Fiona¹²,Kaiko Gerard E.¹²,Board Philip G.³,Keely Simon¹²,Yang Ming¹²^ORCID,Foster Paul S.¹²

Affiliation:

1. Faculty of Health and Medicine School of Biomedical Sciences and Pharmacy University of Newcastle Callaghan NSW Australia

2. Hunter Medical Research Institute New Lambton Heights NSW Australia

3. ACRF Department of Cancer Biology and Therapeutics John Curtin School of Medical Research Australian National University Canberra ACT Australia

Funder

National Health and Medical Research Council

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/cea.13582

Reference43 articles.

1. Distinct TLR-mediated pathways regulate house dust mite–induced allergic disease in the upper and lower airways

2. Cutting Edge: Direct Interaction of TLR4 with NAD(P)H Oxidase 4 Isozyme Is Essential for Lipopolysaccharide-Induced Production of Reactive Oxygen Species and Activation of NF-κB

3. The TLR4 Agonist Immunomax Affects the Phenotype of Mouse Lung Macrophages during Respiratory Syncytial Virus Infection

4. Toll-Like Receptor 4 Is Required for Optimal Development of Th2 Immune Responses: Role of Dendritic Cells

5. TLR2, TLR4 AND MyD88 Mediate Allergic Airway Disease (AAD) and Streptococcus pneumoniae-Induced Suppression of AAD

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