Effects of N-n-butyl Haloperidol Iodide on Myocardial Ischemia/Reperfusion Injury and Egr-1 Expression in Rat-Reference-Cited by-同舟云学术

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Effects of N-n-butyl Haloperidol Iodide on Myocardial Ischemia/Reperfusion Injury and Egr-1 Expression in Rat

Published:2006-06 Issue:6 Volume:38 Page:435-441
ISSN:1672-9145
Container-title:Acta Biochimica et Biophysica Sinica
language:en
Short-container-title:Acta Biochim Biophys Sinica

Author:

ZHANG Yan-Mei,SHI Gang-Gang,TANG Zhao,ZHENG Jin-Hong,LI Wei-Qiu,GUO Fu-Xiao,JIA Qiang-Yong

Publisher

China Science Publishing & Media Ltd.

Subject

General Medicine,Biochemistry,Biophysics

Link

http://academic.oup.com/abbs/article-pdf/38/6/435/984/38-6-435.pdf

Cited by 20 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Temporal changes in Egr-1 and c-fos expression in rat models of myocardial ischemia;Annals of Palliative Medicine;2021-02

2. N-n-Butyl Haloperidol Iodide, a Derivative of the Anti-psychotic Haloperidol, Antagonizes Hypoxia/Reoxygenation Injury by Inhibiting an Egr-1/ROS Positive Feedback Loop in H9c2 Cells;Frontiers in Pharmacology;2018-01-25

3. Characterization of Gene Expression in the Rat Brainstem After Neonatal Hypoxic–Ischemic Injury and Antioxidant Treatment;Molecular Neurobiology;2016-01-25

4. Apelin-13 upregulates Egr-1 expression in rat vascular smooth muscle cells through the PI3K/Akt and PKC signaling pathways;Biochemical and Biophysical Research Communications;2015-12

5. Effect of N-n-butyl haloperidol iodide on ROS/JNK/Egr-1 signaling in H9c2 cells after hypoxia/reoxygenation;Scientific Reports;2015-07-02

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