Haemolysis in sickle cell anaemia: effects of polymorphisms in α-globin gene regulatory elements
Author:
Affiliation:
1. Department of Biostatistics; Boston University School of Public Health; Boston MA USA
2. Department of Medicine; Boston University School of Medicine; Boston MA USA
Funder
National Institutes of Health
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/bjh.15852/fullpdf
Reference8 articles.
1. Effects of α-thalassemia and sickle polymerization tendency on the urine-concentrating defect of individuals with sickle cell trait;Gupta;Journal of Clinical Investigation,1991
2. Genetic dissection of the α-globin super-enhancer in vivo;Hay;Nature Genetics,2016
3. Intravascular hemolysis and the pathophysiology of sickle cell disease;Kato;Journal of Clinical Investigation,2017
4. Genetic determinants of haemolysis in sickle cell anaemia;Milton;British Journal of Haematology,2013
5. The relationship between the severity of hemolysis, clinical manifestations and risk of death in 415 patients with sickle cell anemia in the US and Europe;Nouraie;Haematologica,2013
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2. MZF1 regulates α-globin gene transcription via long-range interactions in erythroid differentiation;Blood Cells, Molecules, and Diseases;2021-03
3. Regulatory Mutation Study in Cases with Unsolved Hypochromic Microcytic Anemia and α-Major Regulatory Element Haplotype Analysis in Iran;Hemoglobin;2021-01-02
4. Sickle cell disease in the era of precision medicine: looking to the future;Expert Review of Precision Medicine and Drug Development;2019-11-02
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