Quantitative proteomic analysis after neuroprotective MyD88 inhibition in the retinal degeneration 10 mouse
Author:
Affiliation:
1. Bascom Palmer Eye Institute University of Miami Miller School of Medicine Miami FL USA
2. Miami Integrative Metabolomics Research Center Miami FL USA
Funder
National Eye Institute
Publisher
Wiley
Subject
Cell Biology,Molecular Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.16893
Reference56 articles.
1. TLR signaling pathways
2. Increased Neuroprotective Microglia and Photoreceptor Survival in the Retina from a Peptide Inhibitor of Myeloid Differentiation Factor 88 (MyD88)
3. Toll-like receptor 3 (TLR3) protects retinal pigmented epithelium (RPE) cells from oxidative stress through a STAT3-dependent mechanism
4. Toll-like receptor 4 inhibitor protects against retinal ganglion cell damage induced by optic nerve crush in mice
5. Photoreceptor Proteins Initiate Microglial Activation via Toll-like Receptor 4 in Retinal Degeneration Mediated by All-trans-retinal
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1. Neuroinflammation in retinitis pigmentosa: Therapies targeting the innate immune system;Frontiers in Immunology;2022-10-27
2. The cytokine IL-27 reduces inflammation and protects photoreceptors in a mouse model of retinal degeneration;Journal of Neuroinflammation;2022-09-05
3. Mass spectrometry‐based retina proteomics;Mass Spectrometry Reviews;2022-06-06
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