Generation of new transgenic <scp>SMARCA4</scp> ‐deficient mouse models results in neuromuscular weakness and paralysis of limbs-Reference-Cited by-同舟云学术

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Generation of new transgenic SMARCA4 ‐deficient mouse models results in neuromuscular weakness and paralysis of limbs

Published:2022-12-29 Issue:3 Volume:33 Page:
ISSN:1015-6305
Container-title:Brain Pathology
language:en
Short-container-title:Brain Pathology

Author:

Neyazi Sina¹²³,Altendorf Lea¹²,Schwetje Dora¹²⁴,Göbel Carolin¹²,Schoof Melanie¹²,Holdhof Dörthe¹²,Schüller Ulrich¹²⁵^ORCID

Affiliation:

1. Department of Pediatric Hematology and Oncology University Medical Center Hamburg‐Eppendorf Hamburg Germany

2. Research Institute Children's Cancer Center Hamburg Hamburg Germany

3. Department of Pediatric Oncology Dana‐Farber Cancer Institute Boston Massachusetts USA

4. Research Group Anatomy, School of Medicine and Health Studies Carl von Ossietzky University Oldenburg Oldenburg Germany

5. Institute of Neuropathology University Medical Center Hamburg‐Eppendorf Hamburg Germany

Funder

Fördergemeinschaft Kinderkrebs‐Zentrum Hamburg

Wilhelm Sander‐Stiftung

Publisher

Wiley

Subject

Neurology (clinical),Pathology and Forensic Medicine,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/bpa.13146

Reference10 articles.

1. The role of chromatin remodeling complexes in Schwann cell development

2. Somatic mutations and single-cell transcriptomes reveal the root of malignant rhabdoid tumours

3. Atypical teratoid/rhabdoid tumors (ATRTs) with SMARCA4 mutation are molecularly distinct from SMARCB1-deficient cases

4. Timing of Smarcb1 and Nf2 inactivation determines schwannoma versus rhabdoid tumor development

5. P0-CreTransgenic Mice for Inactivation of Adhesion Molecules in Schwann Cells

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