Intracellular dialysis disrupts Zn2+ dynamics and enables selective detection of Zn2+ influx in brain slice preparations-Reference-Cited by-同舟云学术

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Intracellular dialysis disrupts Zn2+ dynamics and enables selective detection of Zn2+ influx in brain slice preparations

Published:2013-04-22 Issue:6 Volume:125 Page:822-831
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Aiba Isamu¹,West Adrian K.²,Sheline Christian T.³,Shuttleworth C. William¹

Affiliation:

1. Department of Neurosciences; University of New Mexico; Albuquerque New Mexico USA

2. NeuroRepair Group; Menzies Research Institute; University of Tasmania; Hobart Australia

3. Department of Ophthalmology and the Neuroscience Center of Excellence LSU Health Sciences Center; New Orleans Louisiana USA

Funder

NIH

AHA

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.12246/fullpdf

Reference51 articles.

1. Induction of neuronal apoptosis by thiol oxidation: putative role of intracellular zinc release;Aizenman;J. Neurochem.,2000

2. Ca2+ buffer saturation underlies paired pulse facilitation in calbindin-D28k-containing terminals;Blatow;Neuron,2003

3. Spreading depression and related events are significant sources of neuronal Zn2 + release and accumulation;Carter;J. Cereb. Blood Flow Metab.,2011

4. Protective effects of extracellular glutathione against Zn2+ -induced cell death in vitro and in vivo;Cho;J. Neurosci. Res.,2003

5. Zinc and brain injury;Choi;Annu. Rev. Neurosci.,1998

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