Systemic FasL neutralization increases eosinophilic inflammation in a mouse model of asthma-Reference-Cited by-同舟云学术

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Systemic FasL neutralization increases eosinophilic inflammation in a mouse model of asthma

Published:2011-12-17 Issue:3 Volume:67 Page:328-335
ISSN:0105-4538
Container-title:Allergy
language:en
Short-container-title:

Author:

Sharma S. K.,Almeida F. A.¹,Kierstein S.²,Hortobagyi L.²,Lin T.¹,Larkin A.³,Peterson J.³,Yagita H.⁴,Zangrilli J. G.,Haczku A.²

Affiliation:

1. Division of Pulmonary and Critical Care; Thomas Jefferson University; Philadelphia; PA; USA

2. Pulmonary, Allergy and Critical Care Division; University of Pennsylvania School of Medicine; Philadelphia; PA; USA

3. Center for Translational Medicine; Thomas Jefferson University; Philadelphia; PA; USA

4. Juntendo University School of Medicine; Tokyo; Japan

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1398-9995.2011.02763.x/fullpdf

Reference35 articles.

1. Purification and characterization of the Fas-ligand that induces apoptosis;Suda;J Exp Med,1994

2. Molecular cloning of the Fas ligand, a novel member of the tumor necrosis factor family;Suda;Cell,1993

3. Involvement of ICE-like protease in Fas-mediated apoptosis;Enari;Nature,1995

4. FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis;Chinnaiyan;Cell,1995

5. FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death-inducing signaling complex;Muzio;Cell,1996

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