ABCD1 deletion-induced mitochondrial dysfunction is corrected by SAHA: implication for adrenoleukodystrophy-Reference-Cited by-同舟云学术

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ABCD1 deletion-induced mitochondrial dysfunction is corrected by SAHA: implication for adrenoleukodystrophy

Published:2015-01-13 Issue:3 Volume:133 Page:380-396
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Baarine Mauhamad¹,Beeson Craig²,Singh Avtar¹,Singh Inderjit¹

Affiliation:

1. Department of Pediatrics; Darby Children's Research Institute; Medical University of South Carolina; Charleston South Carolina USA

2. South Carolina College of Pharmacy; Drug Discovery and Biomedical Sciences; Medical University of South Carolina; Charleston South Carolina USA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.12992/fullpdf

Reference98 articles.

1. Reversal of early neurologic and neuroradiologic manifestations of X-linked adrenoleukodystrophy by bone marrow transplantation;Aubourg;N. Engl. J. Med.,1990

2. Peroxisomal and mitochondrial status of two murine oligodendrocytic cell lines (158N, 158JP): potential models for the study of peroxisomal disorders associated with dysmyelination processes;Baarine;J. Neurochem.,2009

3. Evidence of oxidative stress in very long chain fatty acid-treated oligodendrocytes and potentialization of ROS production using RNA interference-directed knockdown of ABCD1 and ACOX1 peroxisomal proteins;Baarine;Neuroscience,2012a

4. Incidence of Abcd1 level on the induction of cell death and organelle dysfunctions triggered by very long chain fatty acids and TNF-alpha on oligodendrocytes and astrocytes;Baarine;Neurotoxicology,2012b

5. X-linked adrenoleukodystrophy: clinical, biochemical and pathogenetic aspects;Berger;Biochim. Biophys. Acta,2006

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