F12-46C/T polymorphism as modifier of the clinical phenotype of hereditary angioedema

Author:

Speletas M.1,Szilágyi Á.2,Csuka D.2,Koutsostathis N.1,Psarros F.3,Moldovan D.4,Magerl M.5,Kompoti M.1,Varga L.2,Maurer M.5,Farkas H.2,Germenis A. E.1

Affiliation:

1. Department of Immunology & Histocompatibility; School of Health Sciences; Faculty of Medicine; University of Thessaly; Larissa Greece

2. 3rd Department of Internal Medicine; Hungarian Angioedema Center; Semmelweis University; Budapest Hungary

3. Department of Allergology; Navy Hospital; Athens Greece

4. Department of Allergy-Immunology; University of Medicine and Pharmacy; Mures County Hospital; Tîrgu Mureș Romania

5. Department of Dermatology and Allergy; Charité - Universitätsmedizin Berlin; Berlin Germany

Funder

Shire Hellas

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Reference19 articles.

1. Overview of hereditary angioedema caused by C1-inhibitor deficiency: assessment and clinical management;Bork;Eur Ann Allergy Clin Immunol,2013

2. Hereditary angioedema;Zuraw;N Engl J Med,2008

3. The pathophysiology of hereditary angioedema;Zuraw;World Allergy Organ J,2010

4. Bradykinin and the pathogenesis of hereditary angioedema;Kaplan;World Allergy Organ J,2011

5. Current update on cellular and molecular mechanisms of hereditary angioedema;Walford;Ann Allergy Asthma Immunol,2014

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