Insecticide resistance mediated by an exon skipping event-Reference-Cited by-同舟云学术

Insecticide resistance mediated by an exon skipping event

Published:2016-11 Issue:22 Volume:25 Page:5692-5704
ISSN:0962-1083
Container-title:Molecular Ecology
language:en
Short-container-title:Mol Ecol

Author:

Berger Madeleine¹²,Puinean Alin Mirel¹,Randall Emma³,Zimmer Christoph T.³,Silva Wellington M.⁴,Bielza Pablo⁵,Field Linda M.¹,Hughes David¹,Mellor Ian²,Hassani‐Pak Keywan¹,Siqueira Herbert A. A.⁴,Williamson Martin S.¹,Bass Chris³

Affiliation:

1. Rothamsted Research Harpenden AL5 2JQ UK

2. School of Life Sciences University of Nottingham Nottingham NG7 2RD UK

3. College of Life and Environmental Sciences Biosciences University of Exeter Penryn Campus Penryn Cornwall TR10 9FE UK

4. Departamento de Agronomia—(Entomologia) Universidade Federal Rural de Pernambuco 52171‐900 Recife PE Brazil

5. Departamento de Producción Vegetal Universidad Politécnica de Cartagena Paseo Alfonso XIII 48 Cartagena 30203 Spain

Funder

Biotechnology and Biological Sciences Research Council

Seventh Framework Programme

Research Executive Agency

Publisher

Wiley

Subject

Genetics,Ecology, Evolution, Behavior and Systematics

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/mec.13882

Reference51 articles.

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2. Spinosad resistance of melon thrips, Thrips palmi, is conferred by G275E mutation in α6 subunit of nicotinic acetylcholine receptor and cytochrome P450 detoxification

3. Gene amplification and microsatellite polymorphism underlie a recent insect host shift

4. Mis-Spliced Transcripts of Nicotinic Acetylcholine Receptor α6 Are Associated with Field Evolved Spinosad Resistance in Plutella xylostella (L.)

5. Incorporation of the β3 Subunit Has a Dominant-Negative Effect on the Function of Recombinant Central-Type Neuronal Nicotinic Receptors

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