Interleukin‐16 aggravates ovalbumin‐induced allergic inflammation by enhancing Th2 and Th17 cytokine production in a mouse model-Reference-Cited by-同舟云学术

Interleukin‐16 aggravates ovalbumin‐induced allergic inflammation by enhancing Th2 and Th17 cytokine production in a mouse model

Published:2019-06-21 Issue:3 Volume:157 Page:257-267
ISSN:0019-2805
Container-title:Immunology
language:en
Short-container-title:Immunology

Author:

Li Chunxia¹,Dai Jun¹,Dong Guanjun¹,Ma Qun¹,Li Zhihua¹,Zhang Hui¹,Yan Fenglian¹,Zhang Junfeng¹^ORCID,Wang Bo¹,Shi Hui¹,Zhu Yuzhen¹,Yao Xiaoying¹,Si Chuanping¹,Xiong Huabao²^ORCID

Affiliation:

1. Institute of Immunology and Molecular Medicine Jining Medical University Shandong China

2. Department of Medicine Precision Immunology Institute Icahn School of Medicine at Mount Sinai New York NY USA

Funder

National Natural Science Foundation of China

Jining Medical University

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/imm.13068

Reference43 articles.

1. Allergic diseases and asthma

2. Mouse models of allergic asthma: acute and chronic allergen challenge

3. A Murine IL-4 Receptor Antagonist That Inhibits IL-4- and IL-13-Induced Responses Prevents Antigen-Induced Airway Eosinophilia and Airway Hyperresponsiveness

4. The Absence of Interleukin 9 Does Not Affect the Development of Allergen-induced Pulmonary Inflammation nor Airway Hyperreactivity

5. Resolution of Bronchial Hyperresponsiveness and Pulmonary Inflammation Is Associated with IL-3 and Tissue Leukocyte Apoptosis

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