Myostatin dysfunction is associated with reduction in overload induced hypertrophy of soleus muscle in mice

Author:

Minderis P.1,Kilikevicius A.1,Baltusnikas J.1,Alhindi Y.2,Venckunas T.1,Bunger L.3,Lionikas A.12,Ratkevicius A.12

Affiliation:

1. Institute of Sport Science and Innovations; Lithuanian Sports University; Kaunas Lithuania

2. School of Medical Sciences; College of Life Sciences and Medicine; University of Aberdeen; Aberdeen Scotland UK

3. Animal Breeding and Genetics, Animal and Veterinary Sciences group; Scotland's Rural College (SRUC); Edinburgh UK

Funder

Marie Curie International Reintegration Grant

Lietuvos Mokslo Taryba

Ministry of Higher Education of Saudi Arabia

Publisher

Wiley

Subject

Physical Therapy, Sports Therapy and Rehabilitation,Orthopedics and Sports Medicine

Reference41 articles.

1. Lack of myostatin results in excessive muscle growth but impaired force generation;Amthor;Proc Natl Acad Sci U S A,2007

2. Phosphorylation of p70(S6k) correlates with increased skeletal muscle mass following resistance exercise;Baar;Am J Physiol,1999

3. Myostatin dysfunction impairs force generation in extensor digitorum longus muscle and increases exercise-induced protein efflux from extensor digitorum longus and soleus muscles;Baltusnikas;Appl Physiol Nutr Metab,2015a

4. Regenerated soleus muscle shows reduced creatine kinase efflux after contractile activity in vitro;Baltusnikas;Appl Physiol Nutr Metab,2015b

5. Rapid determination of myosin heavy chain expression in rat, mouse, and human skeletal muscle using multicolor immunofluorescence analysis;Bloemberg;PLoS ONE,2012

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