Persistent expression of interleukin‐17 and downstream effector cytokines in recalcitrant psoriatic lesions after ustekinumab treatment
Author:
Affiliation:
1. Department of Dermatology Seoul National University College of Medicine and Seoul National University Bundang Hospital Seongnam Korea
2. Department of Pathology Seoul National University Bundang Hospital Seongnam Korea
Publisher
Wiley
Subject
Dermatology,General Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/1346-8138.15822
Reference27 articles.
1. Psoriasis
2. Interleukin 17A: Toward a new understanding of psoriasis pathogenesis
3. The Interleukin-23/Interleukin-17 Axis Links Adaptive and Innate Immunity in Psoriasis
4. Increased Expression of Interleukin 23 p19 and p40 in Lesional Skin of Patients with Psoriasis Vulgaris
5. Expression of Th17 cytokines in skin lesions of patients with psoriasis
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1. Brodalumab Versus Guselkumab in Patients with Moderate-to-Severe Psoriasis with an Inadequate Response to Ustekinumab: A Randomized, Multicenter, Double-Blind Phase 4 Trial (COBRA);Dermatology and Therapy;2024-02
2. Could Targeted Pharmacotherapies Exert a “Disease Modification Effect” in Patients with Chronic Plaque Psoriasis?;International Journal of Molecular Sciences;2022-10-25
3. Interleukine-17 Modulates Neurogenesis and Behavior Following Exposure to Trauma in Mice;Cells;2022-01-20
4. Epidermal Hyperproliferation With Less Prominent Dermal Inflammation Is the Unique Histopathological Feature of the Refractory Lesions in Psoriasis Treated With Ustekinumab;The American Journal of Dermatopathology;2021-11-02
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