Ezrin contributes to impaired podocyte migration and adhesion caused by advanced glycation end products
Author:
Affiliation:
1. Department of Medicine (Alfred); Monash University; Melbourne Victoria Australia
2. Department of Endocrinology and Diabetes; Alfred Hospital; Melbourne Victoria Australia
Funder
Diabetes Australia Research Trust
Publisher
Wiley
Subject
Nephrology,General Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/nep.12526/fullpdf
Reference34 articles.
1. Mechanisms of diabetic complications;Forbes;Physiol. Rev.,2013
2. Insulin signaling to the glomerular podocyte is critical for normal kidney function;Welsh;Cell Metab.,2010
3. Essential role of integrin-linked kinase in podocyte biology: Bridging the integrin and slit diaphragm signaling;Dai;J. Am. Soc. Nephrol.,2006
4. Emerging role for ERM proteins in cell adhesion and migration;Arpin;Cell Adh. Migr.,2011
5. Loss of glomerular foot processes is associated with uncoupling of podocalyxin from the actin cytoskeleton;Takeda;J. Clin. Invest.,2001
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