High glucose induced calcium overload via impairment of SERCA / PLN pathway and mitochondrial dysfunction leads to oxidative stress in H9c2 cells and amelioration with ferulic acid-Reference-Cited by-同舟云学术

High glucose induced calcium overload via impairment of SERCA / PLN pathway and mitochondrial dysfunction leads to oxidative stress in H9c2 cells and amelioration with ferulic acid

Published:2019-03-18 Issue:4 Volume:33 Page:412-425
ISSN:0767-3981
Container-title:Fundamental & Clinical Pharmacology
language:en
Short-container-title:Fundam Clin Pharmacol

Author:

Salin Raj Palayyan¹²,Swapna Sasi U. S.¹²,Raghu Kozhiparambil G.¹²^ORCID

Affiliation:

1. Biochemistry and Molecular Mechanism Laboratory Agro‐Processing and Technology Division CSIR ‐ National Institute for Interdisciplinary Science and Technology (NIIST) Thiruvananthapuram 695019 Kerala India

2. Academy of Scientific and Innovative Research (AcSIR) CSIR‐HRDC Campus Ghaziabad Uttar Pradesh 201 002 India

Publisher

Wiley

Subject

Pharmacology (medical),Pharmacology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/fcp.12452

Reference53 articles.

1. A new insight of mechanisms, diagnosis and treatment of diabetic cardiomyopathy

2. Diabetic Cardiovascular Disease Induced by Oxidative Stress

3. High glucose-induced Ca2+ overload and oxidative stress contribute to apoptosis of cardiac cells through mitochondrial dependent and independent pathways

4. Facilitated Ca2+ homeostasis and attenuated myocardial autophagy contribute to alleviation of diabetic cardiomyopathy after bariatric surgery

5. Chemistry, natural sources, dietary intake and pharmacokinetic properties of ferulic acid: A review

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