Necessity of protein kinase C activity for maintenance of acetylcholine receptor function at snake twitch fibre endplates
Author:
Publisher
Wiley
Subject
Pharmacology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1476-5381.1995.tb13245.x/fullpdf
Reference32 articles.
1. Cytochalasin modulation of nicotinic cholinergic receptor expression and muscarinic receptor function in human TE671/RD cells: a possible functional role of the cytoskeleton;BENCHERIF;J. Neurochem.,1993
2. Comparison of cholinergic activation and desensitization at snake twitch and slow muscle fibre end-plates;CONNOR;J. Physiol.,1984
3. Direct measurement of the concentration- and time-dependent open probability of the nicotinic acetylcholine receptor channel;DILGER;Biophys. J.,1990
4. Desensitization of acetylcholine receptors in BC3H-1 cells;DILGER;Pflügers Arch.,1992
5. Two types of nicotinic acetylcholine receptor channels at slow fibre end-plates of the garter snake;DIONNE;J. Physiol.,1988
Cited by 4 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Activation of protein kinase C increases acetylcholine release from frog motor nerves by a direct action on L-type Ca2+ channels and apparently not by depolarisation of the terminal;Neuroscience;2001-07
2. Activation of the protein phosphatase calcineurin during carbachol exposure decreases the extent of recovery from end-plate desensitization;Journal of Neurophysiology;1996-12-01
3. POSTER COMMUNICATIONS;British Journal of Pharmacology;1996-04
4. Requirement of a colchicine-sensitive component of the cytoskeleton for acetylcholine receptor recovery;British Journal of Pharmacology;1995-01
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