Negative inotropic activity of antilaminin IgG: participation of cholinergic mechanisms
Author:
Publisher
Wiley
Subject
Pharmacology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1476-5381.1989.tb11964.x/fullpdf
Reference23 articles.
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2. Nerve growth factor, laminin and fibronectin promote neurite growth in human fetal sensory ganglia cultures;Evercooren;J. Neurosc. Res,1982
3. Lymphocytes sensitize rat isolated atria to the inotropic and chronotropic effect of sodium arachidonate;Borda;Br. J. Pharmacol,1984a
4. A circulating IgG in Chagas’ disease which bind to beta adrenoceptors of myocardium and modulates their activity;Borda;Clin Exp. Immunol,1984b
5. Role of beta adrenoceptors in the biological effect of autoimmune orchitis spleen cells;Borda;Can. J. Physiol. Pharmacol,1986
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1. Modulation of cardiac physiology by an anti‐Trypanosoma cruzi monoclonal antibody after interaction with myocardium;The FASEB Journal;1995-11
2. Participation of cytoskeleton in the effect of antilaminin IgG on cardiac cholinoceptors;British Journal of Pharmacology;1994-01
3. Ab initio study of the electronic properties of potential antagonists of the glycine receptor: 1. Transition state of the 2-pyridone · H2O/2-hydroxypyridine · H2O tautomeric equilibrium;International Journal of Quantum Chemistry;1993
4. Antilaminin IgG triggers the murine atria phosphoinositide hydrolysis through muscarinic receptor stimulation;International Journal of Immunopharmacology;1992-11
5. Antilaminin IgG releases TXB2 through activation of the cholinergic system;Prostaglandins, Leukotrienes and Essential Fatty Acids;1990-10
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