Prevention by the NMDA receptor antagonist, MK801 of neuronal loss produced by tetanus toxin in the rat hippocampus
Author:
Publisher
Wiley
Subject
Pharmacology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1476-5381.1990.tb14156.x/fullpdf
Reference27 articles.
1. Tetanus toxin produces neuronal loss and a reduction in GABAA but not GABAB binding sites in rat hippocampus;BAGETTA;Neurosci. Lett,1990
2. Elevation of the extracellular concentration of glutamate and aspartate in rat hippocampus during transient cerebral ischaemia monitored by intracerebral microdialysis;BENEVISTE;J. Neurochem,1984
3. Differential effects of tetanus toxin on inhibitory and excitatory synaptic transmission in mammalian spinal cord neurones in culture; a presynaptic locus of action for tetanus toxin;BERGEY;J. Neurophysiol,1987
4. Selective depression of synaptic transmission of tetanus toxin: a comparative study on hippocampal and neostriatal slices;CALABRESI;Neuroscience,1989
5. Anticonvulsant activity of (+)-5-methyl-10,11-olihydro-5H-dibenzo-[a, d]-cyclohepten-5,10-imine (MK801), a substance with potent anticonvulsant, central sympathomimetic and apparent anxiolytic properties;CLINESCHMIDT;Drug Dev. Res,1982
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