P2X7 activation enhances skeletal muscle metabolism and regeneration in SOD1G93A mouse model of amyotrophic lateral sclerosis-Reference-Cited by-同舟云学术

P2X7 activation enhances skeletal muscle metabolism and regeneration in SOD1G93A mouse model of amyotrophic lateral sclerosis

Published:2019-08-18 Issue:2 Volume:30 Page:272-282
ISSN:1015-6305
Container-title:Brain Pathology
language:en
Short-container-title:Brain Pathol

Author:

Fabbrizio Paola¹,Apolloni Savina²,Bianchi Andrea²,Salvatori Illari²,Valle Cristiana²³,Lanzuolo Chiara²⁴,Bendotti Caterina¹,Nardo Giovanni¹^ORCID,Volonté Cinzia²⁵^ORCID

Affiliation:

1. Laboratory of Molecular Neurobiology, Department of Neuroscience Istituto di Ricerche Farmacologiche Mario Negri IRCCS Milan Italy

2. IRCCS Fondazione Santa Lucia Rome Italy

3. National Research Council, Institute of Translational Pharmacology Rome Italy

4. National Research Council, Institute of Biomedical Technologies Milan Italy

5. National Research Council, Institute for Systems Analysis and Computer Science Rome Italy

Funder

Agenzia di Ricerca per la Sclerosi Laterale Amiotrofica

Publisher

Wiley

Subject

Neurology (clinical),Pathology and Forensic Medicine,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/bpa.12774

Reference51 articles.

1. Molecular circuitry of stem cell fate in skeletal muscle regeneration, ageing and disease

2. Ablation of P2X7 receptor exacerbates gliosis and motoneuron death in the SOD1-G93A mouse model of amyotrophic lateral sclerosis

3. Spinal cord pathology is ameliorated by P2X7 antagonism in SOD1-G93A mouse model of amyotrophic lateral sclerosis

4. The NADPH Oxidase Pathway Is Dysregulated by the P2X7 Receptor in the SOD1-G93A Microglia Model of Amyotrophic Lateral Sclerosis

5. P2X7 antagonism using Brilliant Blue G reduces body weight loss and prolongs survival in female SOD1G93Aamyotrophic lateral sclerosis mice

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