Impaired renal HCO 3 − secretion in CFTR deficient mice causes metabolic alkalosis during chronic base‐loading

Author:

Berg Peder1,Svendsen Samuel L.1,Hoang Thi Thuy Linh1,Praetorius Helle A.1,Sorensen Mads V.1,Leipziger Jens12ORCID

Affiliation:

1. Department of Biomedicine, Physiology Faculty of Health Aarhus University Aarhus C Denmark

2. Aarhus Institute of Advanced Studies Aarhus University Aarhus C Denmark

Funder

Fonden til Lægevidenskabens Fremme

Helga og Peter Kornings Fond

Det Frie Forskningsråd

Publisher

Wiley

Subject

Physiology

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1. Secretin: a hormone for HCO3− homeostasis;Pflügers Archiv - European Journal of Physiology;2024-01-15

2. Novel functions of the anion exchanger AE4 (SLC4A9);Pflügers Archiv - European Journal of Physiology;2024-01-09

3. Trimethoprim inhibits renal H+-K+-ATPase in states of K+ depletion;American Journal of Physiology-Renal Physiology;2024-01-01

4. Pendrin: linking acid base to blood pressure;Pflügers Archiv - European Journal of Physiology;2023-12-19

5. The B1 H+-ATPase (Atp6v1b1) Subunit in Non–Type A Intercalated Cells is Required for Driving Pendrin Activity and the Renal Defense Against Alkalosis;Journal of the American Society of Nephrology;2023-11-22

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