Oligodendrocyte‐specific deletion of FGFR2 ameliorates MOG 35‐55 ‐induced EAE through ERK and Akt signalling-Reference-Cited by-同舟云学术

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Oligodendrocyte‐specific deletion of FGFR2 ameliorates MOG 35‐55 ‐induced EAE through ERK and Akt signalling

Published:2021-01-04 Issue:2 Volume:31 Page:297-311
ISSN:1015-6305
Container-title:Brain Pathology
language:en
Short-container-title:Brain Pathol.

Author:

Kamali Salar¹,Rajendran Ranjithkumar¹,Stadelmann Christine²,Karnati Srikanth³,Rajendran Vinothkumar¹,Giraldo‐Velasquez Mario⁴,Berghoff Martin¹^ORCID

Affiliation:

1. Department of Neurology University of Giessen Giessen Germany

2. Institute of Neuropathology University Medical Center Göttingen Göttingen Germany

3. Institute of Anatomy and Cell Biology University of Würzburg Würzburg Germany

4. Department of Neurology Sozialstiftung Bamberg Bamberg Germany

Publisher

Wiley

Subject

Neurology (clinical),Pathology and Forensic Medicine,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/bpa.12916

Reference39 articles.

1. Multiple sclerosis: experimental models and reality

2. Oligodendrocyte regeneration: Its significance in myelin replacement and neuroprotection in multiple sclerosis

3. Central nervous system rather than immune cell-derived BDNF mediates axonal protective effects early in autoimmune demyelination

4. Functional role of brain-derived neurotrophic factor in neuroprotective autoimmunity: therapeutic implications in a model of multiple sclerosis

5. LINGO-1 antagonist promotes spinal cord remyelination and axonal integrity in MOG-induced experimental autoimmune encephalomyelitis

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1. Functional Role of DDR1 in Oligodendrocyte Signaling Mechanism in Association with Myelination and Remyelination Process in the Central Nerve System;Journal of Physiological Investigation;2024-07

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3. Immunopathogenesis of multiple sclerosis: molecular and cellular mechanisms and new immunotherapeutic approaches;Immunopharmacology and Immunotoxicology;2024-04-18

4. Fibroblast growth factor receptor inhibitors mitigate the neuropathogenicity of Borrelia burgdorferi or its remnants ex vivo;Frontiers in Immunology;2024-04-04

5. Fgf22 and Fgfr2b are required for neurogenesis and gliogenesis in the zebrafish forebrain;Biochemical and Biophysical Research Communications;2023-11

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