HbA 2 induction: the merit of pancellularity in sickle cell disease
Author:
Affiliation:
1. Department of Medicine Division of Hematology/Oncology Center of Excellence for Sickle Cell Disease Center for Regenerative Medicine Genome Science InstituteBoston University School of Medicine and Boston Medical Center Boston MA USA
Publisher
Wiley
Subject
Hematology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/bjh.17562
Reference16 articles.
1. Structural bases of the inhibitory effects of hemoglobin F and hemoglobin A2 on the polymerization of hemoglobin S.
2. Sparing effect of hemoglobin F and hemoglobin A2 on the polymerization of hemoglobin S at physiologic ligand saturations.
3. Activation of delta-globin gene expression by erythroid Krupple-like factor: a potential approach for gene therapy of sickle cell disease
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3. Fetal hemoglobin per erythrocyte ( HbF /F‐cell) after gene therapy for sickle cell anemia;American Journal of Hematology;2022-11-30
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