Traumatic Brain Injury Elevates the Alzheimer's Amyloid Peptide Abeta42 in Human CSF: A Possible Role for Nerve Cell Injury-Reference-Cited by-同舟云学术

Traumatic Brain Injury Elevates the Alzheimer's Amyloid Peptide Abeta42 in Human CSF: A Possible Role for Nerve Cell Injury

Published:2000-04 Issue:1 VASCULAR FACT Volume:903 Page:118-122
ISSN:0077-8923
Container-title:Annals of the New York Academy of Sciences
language:en
Short-container-title:Annals NY Acad Sci

Author:

EMMERLING M. R.,MORGANTI-KOSSMANN M. C.,KOSSMANN T.,STAHEL P. F.,WATSON M. D.,EVANS L. M.,MEHTA P. D.,SPIEGEL K.,KUO Y.-M.,ROHER A. E.,RABY C. A.

Publisher

Wiley

Subject

History and Philosophy of Science,General Biochemistry, Genetics and Molecular Biology,General Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1749-6632.2000.tb06357.x/fullpdf

Reference18 articles.

1. The cell biology of β-amyloid precursor protein and presenilin in Alzheimer's disease

2. Amyloid β-Protein, APOE Genotype and Head Injury

3. The role of the complement system in traumatic brain injury

4. 4Emmerling, M.R., S. Gracon & A.E. Roher. 1999. Towards a Unifying Hypothesis of Alzheimer's Disease: Pathogenesis and Pathophysiology.: 33-53. Martin Dunitz Ltd. London.

5. INTRATHECAL AND SERUM INTERLEUKIN-6 AND THE ACUTE-PHASE RESPONSE IN PATIENTS WITH SEVERE TRAUMATIC BRAIN INJURIES

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