Pre‐existing inflammation reduces the response to contact allergens in Tmem79‐deficient mice-Reference-Cited by-同舟云学术

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Pre‐existing inflammation reduces the response to contact allergens in Tmem79‐deficient mice

Published:2024-02-17 Issue:7 Volume:79 Page:1978-1981
ISSN:0105-4538
Container-title:Allergy
language:en
Short-container-title:Allergy

Author:

Jee Mia Hamilton¹²^ORCID,Funch Anders Boutrup¹²^ORCID,Weber Julie Friis¹^ORCID,Yeung Kelvin¹^ORCID,Mraz Veronika¹^ORCID,Gadsbøll Anne‐Sofie Østergaard¹^ORCID,Song Tinghuan¹^ORCID,Woetmann Anders¹^ORCID,Ødum Niels¹^ORCID,Johansen Jeanne Duus²^ORCID,Geisler Carsten¹^ORCID,Bonefeld Charlotte Menné¹^ORCID

Affiliation:

1. Department of Immunology and Microbiology, LEO Foundation Skin Immunology Research Center, Faculty of Health and Medical Sciences University of Copenhagen Copenhagen Denmark

2. Department of Dermato‐Allergology, National Allergy Research Centre Copenhagen University Hospital Herlev‐Gentofte Hellerup Denmark

Publisher

Wiley

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/all.16053

Reference6 articles.

1. Association between atopic dermatitis and contact sensitization: A systematic review and meta-analysis

2. Contact Sensitivity to Dinitrochlorobenzene Is Impaired in Atopic Subjects

3. CD8 + tissue‐resident memory T cells recruit neutrophils that are essential for flare‐ups in contact dermatitis

4. Tmem79/Matt is the matted mouse gene and is a predisposing gene for atopic dermatitis in human subjects

5. Dysregulated skin barrier function in Tmem79 mutant mice promotes IL‐17A‐dependent spontaneous skin and lung inflammation

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