JNK modifies neuronal metabolism to promote proteostasis and longevity-Reference-Cited by-同舟云学术

JNK modifies neuronal metabolism to promote proteostasis and longevity

Published:2019-02-27 Issue:3 Volume:18 Page:
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Wang Lifen¹²,Davis Sonnet S.¹,Borch Jensen Martin¹,Rodriguez‐Fernandez Imilce A.¹,Apaydin Cagsar¹,Juhasz Gabor³⁴,Gibson Bradford W.¹,Schilling Birgit¹,Ramanathan Arvind¹,Ghaemmaghami Sina⁵,Jasper Heinrich¹²

Affiliation:

1. The Buck Institute for Research on Aging Novato California

2. Genentech Inc. South San Francisco California

3. Department of Anatomy, Cell and Developmental Biology Eotvos Lorand University Budapest Hungary

4. Institute of Genetics Biological Research Center of the Hungarian Academy of Sciences Szeged Hungary

5. Department of Biology University of Rochester Rochester New York

Funder

Magyar Tudományos Akadémia

National Institute of General Medical Sciences

National Institute on Aging

National Center for Research Resources

National Science Foundation

Publisher

Wiley

Subject

Cell Biology,Aging

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.12849

Reference62 articles.

1. Amyloid-binding compounds maintain protein homeostasis during ageing and extend lifespan

2. Age- and diet-associated metabolome remodeling characterizes the aging process driven by damage accumulation

3. Enhanced Neuronal Glucose Transporter Expression Reveals Metabolic Choice in a HD Drosophila Model

4. Increased energy metabolism rescues glia-induced pathology in a Drosophila model of Huntington's disease

5. Regulation of Drosophila lifespan by JNK signaling

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