A dual role for Integrin α6β4 in modulating hereditary neuropathy with liability to pressure palsies-Reference-Cited by-同舟云学术

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A dual role for Integrin α6β4 in modulating hereditary neuropathy with liability to pressure palsies

Published:2018-02-13 Issue:3 Volume:145 Page:245-257
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Poitelon Yannick¹²^ORCID,Matafora Vittoria³^ORCID,Silvestri Nicholas⁴,Zambroni Desirée³^ORCID,McGarry Claire²,Serghany Nora²,Rush Thomas²,Vizzuso Domenica¹³,Court Felipe A.³⁵^ORCID,Bachi Angela³^ORCID,Wrabetz Lawrence¹²³⁴^ORCID,Feltri Maria Laura¹²³⁴^ORCID

Affiliation:

1. Hunter James Kelly Research Institute; University at Buffalo; Buffalo New York USA

2. Department of Biochemistry; University at Buffalo; Buffalo New York USA

3. Division of Genetics and Cell Biology; San Raffaele Hospital; Milan Italy

4. Department of Neurology; University at Buffalo; Buffalo New York USA

5. Center for Integrative Biology; Universidad Mayor de Chile; Santiago Chile

Funder

Fondazione Telethon

National Institutes of Health

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.14295/fullpdf

Reference50 articles.

1. Hypermyelination and demyelinating peripheral neuropathy in Pmp22-deficient mice;Adlkofer;Nat. Genet.,1995

2. Heterozygous peripheral myelin protein 22-deficient mice are affected by a progressive demyelinating tomaculous neuropathy;Adlkofer;J. Neurosci.,1997

3. Peripheral myelin protein 22 is in complex with alpha6beta4 integrin, and its absence alters the Schwann cell basal lamina;Amici;J. Neurosci.,2006

4. Developmental abnormalities in the nerves of peripheral myelin protein 22-deficient mice;Amici;J. Neurosci. Res.,2007

5. Conduction block in PMP22 deficiency;Bai;J. Neurosci.,2010

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