Antisense‐induced knockdown of cAMP response element‐binding protein downregulates Per1 gene expression in the shell region of nucleus accumbens resulting in reduced alcohol consumption in mice-Reference-Cited by-同舟云学术

Antisense‐induced knockdown of cAMP response element‐binding protein downregulates Per1 gene expression in the shell region of nucleus accumbens resulting in reduced alcohol consumption in mice

Published:2021-08-23 Issue:10 Volume:45 Page:1940-1949
ISSN:0145-6008
Container-title:Alcoholism: Clinical and Experimental Research
language:en
Short-container-title:Alcohol Clin Exp Res

Author:

Sharma Rishi¹,Mishra Vaibhav¹,Parikh Meet¹,Soni Anshul¹,Sahota Pradeep¹,Thakkar Mahesh¹^ORCID

Affiliation:

1. Department of Neurology Harry S. Truman Memorial Veterans Hospital University of Missouri Columbia Missouri USA

Publisher

Wiley

Subject

Psychiatry and Mental health,Toxicology,Medicine (miscellaneous)

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acer.14687

Reference46 articles.

1. Basic local alignment search tool

2. Expression of the Nuclear Receptor Coactivator, cAMP Response Element-Binding Protein, Is Sexually Dimorphic and Modulates Sexual Differentiation of Neonatal Rat Brain

3. Cannabinoid-1 receptor neutral antagonist reduces binge-like alcohol consumption and alcohol-induced accumbal dopaminergic signaling

4. Dopaminergic Regulation of Striatal Interneurons in Reward and Addiction: Focus on Alcohol

5. cAMP-dependent Protein Kinase Type I Regulates Ethanol-induced cAMP Response Element-mediated Gene Expression via Activation of CREB-binding Protein and Inhibition of MAPK

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2. Cholinergic interneurons in the shell region of the nucleus accumbens regulate binge alcohol consumption: A chemogenetic and genetic lesion study;Alcohol, Clinical and Experimental Research;2024-03-29

3. Antisense‐induced downregulation of major circadian genes modulates the expression of histone deacetylase‐2 (HDAC‐2) and CREB‐binding protein (CBP) in the medial shell region of nucleus accumbens of mice exposed to chronic excessive alcohol consumption;Journal of Neurochemistry;2021-12-05