Severe learning deficits of IRSp53 mutant mice are caused by altered NMDA receptor-dependent signal transduction-Reference-Cited by-同舟云学术

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Severe learning deficits of IRSp53 mutant mice are caused by altered NMDA receptor-dependent signal transduction

Published:2015-11-30 Issue:4 Volume:136 Page:752-763
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Bobsin Kristin¹,Kreienkamp Hans-Jürgen¹

Affiliation:

1. Institut für Humangenetik; Universitätsklinikum Hamburg-Eppendorf; Hamburg Germany

Funder

Deutsche Forschungsgemeinschaft

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.13428/fullpdf

Reference45 articles.

1. The insulin receptor tyrosine kinase substrate p58/53 and the insulin receptor are components of CNS synapses;Abbott;J. Neurosci.,1999

2. Dorsal hippocampus and classical fear conditioning to tone and context in rats: Effects of local NMDA-receptor blockade and stimulation;Bast;Hippocampus,2003

3. The puzzle box as a simple and efficient behavioral test for exploring impairments of general cognition and executive functions in mouse models of schizophrenia;Ben Abdallah;Exp. Neurol.,2011

4. Context preexposure prevents forgetting of a contextual fear memory: implication for regional changes in brain activation patterns associated with recent and remote memory tests;Biedenkapp;Learn Mem.,2007

5. ProSAP/Shank postsynaptic density proteins interact with insulin receptor tyrosine kinase substrate IRSp53;Bockmann;J. Neurochem.,2002

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