Activation of Elk‐1 participates as a neuroprotective compensatory mechanism in models of Huntington’s disease-Reference-Cited by-同舟云学术

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Activation of Elk‐1 participates as a neuroprotective compensatory mechanism in models of Huntington’s disease

Published:2012-03-28 Issue:4 Volume:121 Page:639-648
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:Journal of Neurochemistry

Author:

Anglada‐Huguet Marta¹²³,Giralt Albert¹²³,Perez‐Navarro Esther¹²³,Alberch Jordi¹²³,Xifró Xavier¹²³⁴

Affiliation:

1. Departament de Biologia Cel·lular, Immunologia i Neurociències, Facultat de Medicina, Universitat de Barcelona, Barcelona, Spain

2. Centro de Investigación Biomédica en Red sobre Enfermedades Neurodegenerativas (CIBERNED), Spain

3. Institut d’Investigacions Biomèdiques August Pi i Sunyer (IDIBAPS), Barcelona, Spain

4. Departament de Ciències Mèdiques, Facultat de Medicina, Universitat de Girona, Spain

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/j.1471-4159.2012.07711.x

Reference50 articles.

1. Mutant huntingtin alters MAPK signaling pathways in PC12 and striatal cells: ERK1/2 protects against mutant huntingtin-associated toxicity

2. Elk-1 a Transcription Factor with Multiple Facets in the Brain

3. Overlapping promoter targeting by Elk-1 and other divergent ETS-domain transcription factor family members

4. Ets ternary complex transcription factors

5. Expression of Brain-Derived Neurotrophic Factor in Cortical Neurons Is Regulated by Striatal Target Area

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