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Inherited and acquired interactions between ACHE and PON1 polymorphisms modulate plasma acetylcholinesterase and paraoxonase activities

  • Published:2005-02-04 Issue:5 Volume:92 Page:1216-1227
  • ISSN:0022-3042
  • Container-title:Journal of Neurochemistry
  • language:en
  • Short-container-title:

Author:

Bryk Boris,BenMoyal-Segal Liat,Podoly Erez,Livnah Oded,Eisenkraft Arik,Luria Shai,Cohen Amir,Yehezkelli Yoav,Hourvitz Ariel,Soreq Hermona

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Reference55 articles.

1. Paraoxonase inhibits high-density lipoprotein oxidation and preserves its functions. A possible peroxidative role for paraoxonase;Aviram;J. Clin. Invest.,1998

2. Mutation at codon 322 in the human acetylcholinesterase (ACHE) gene accounts for YT blood group polymorphism;Bartels;Am. J. Hum. Genet.,1993

3. In utero pesticide exposure, maternal paraoxonase activity, and head circumference;Berkowitz;Environ. Health Perspect.,2004

4. The role of readthrough acetylcholinesterase in the pathophysiology of myasthenia gravis;Brenner;Faseb J.,2003

5. Effects of 5′ regulatory-region polymorphisms on paraoxonase-gene (PON1) expression;Brophy;Am. J. Hum. Genet.,2001
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