Identification and characterization of a de novo partial trisomy 10p by comparative genomic hybridization (CGH)
Author:
Publisher
Wiley
Subject
Genetics (clinical),Genetics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1034/j.1399-0004.1998.5440412.x/fullpdf
Reference23 articles.
1. Characterization of familial partial 10p trisomy by chromosomal microdissection, FISH, and microsatellite dosage analysis;Stone;Hum Genet,1996
2. Comparative genomic hybridization for molecular cytogentic analysis of solid tumors;Kallioniemi;Science,1992
3. Detection of complete and partial chromosome gains and losses by comparative genomic in situ hybridization;Manoir;Hum Genet,1993
4. In vivo amplification of the androgen receptor gene and progression of human prostate cancer;Visakorpi;Nat Genet,1995
5. Comparative genomic hybridization in clinical cytogenetics;Bryndorf;Am J Hum Genet,1995
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2. Subtelomeric 6.7 Mb trisomy 10p and 5.6 Mb monosomy 21q detected by FISH and array-CGH in three related patients;American Journal of Medical Genetics Part A;2012-03-09
3. Identification of genomic loci contributing to agenesis of the corpus callosum;American Journal of Medical Genetics Part A;2010-08-03
4. Detection of subtelomeric chromosomal rearrangements in idiopathic mental retardation;Orvosi Hetilap;2010-07-01
5. Familial translocation t(3;10) (p26.3;p12.31) leading to trisomy 10p12.31 → pter and monosomy 3p26.3 → pter in seven members;American Journal of Medical Genetics Part A;2008-12-15
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