Neonatal Hyperoxia Exposure Disrupts Axon-Oligodendrocyte Integrity in the Subcortical White Matter
Author:
Publisher
Society for Neuroscience
Subject
General Neuroscience
Cited by 65 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Timed fetal inflammation and postnatal hypoxia cause cortical white matter injury, interneuron imbalances, and behavioral deficits in a double-hit rat model of encephalopathy of prematurity;Brain, Behavior, & Immunity - Health;2024-10
2. Mechanistic advances of hyperoxia-induced immature brain injury;Heliyon;2024-05
3. A review of the effects of early postnatal hyperoxia exposure on the immature brain;Experimental Neurology;2023-12
4. Association between bronchopulmonary dysplasia and death or neurodevelopmental impairment at 3 years in preterm infants without severe brain injury;Frontiers in Neurology;2023-11-15
5. Key roles of glial cells in the encephalopathy of prematurity;Glia;2023-11
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