CaMKIIα Mediates the Effect of IL-17 To Promote Ongoing Spontaneous and Evoked Pain in Multiple Sclerosis
Author:
Publisher
Society for Neuroscience
Subject
General Neuroscience
Cited by 14 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Mechanisms underlying the beneficial effects of physical exercise on multiple sclerosis: focus on immune cells;Frontiers in Immunology;2023-09-29
2. Spinal Sirtuin 3 Contributes to Electroacupuncture Analgesia in Mice With Chronic Constriction Injury–Induced Neuropathic Pain;Neuromodulation: Technology at the Neural Interface;2023-04
3. Neuroinflammation in the Pathogenesis of Neuropathic Pain Syndrome;Neuroscience and Behavioral Physiology;2023-01
4. Involvement of TLR2–TLR4, NLRP3, and IL-17 in pain induced by a novel Sprague-Dawley rat model of experimental autoimmune encephalomyelitis;Frontiers in Pain Research;2022-09-13
5. Activation of the N-methyl-D-aspartate receptor contributes to orofacial neuropathic and inflammatory allodynia by facilitating calcium-calmodulin-dependent protein kinase II phosphorylation in mice;Brain Research Bulletin;2022-07
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