Defective Fatty Acid Uptake in the Spontaneously Hypertensive Rat Is a Primary Determinant of Altered Glucose Metabolism, Hyperinsulinemia, and Myocardial Hypertrophy-Reference-Cited by-同舟云学术

Defective Fatty Acid Uptake in the Spontaneously Hypertensive Rat Is a Primary Determinant of Altered Glucose Metabolism, Hyperinsulinemia, and Myocardial Hypertrophy

Published:2001-06 Issue:26 Volume:276 Page:23661-23666
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Hajri Tahar,Ibrahimi Azeddine,Coburn Chris T.,Knapp F.F.,Kurtz Ted,Pravenec Michael,Abumrad Nada A.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference35 articles.

1. Quantitative trait loci for cellular defects in glucose and fatty acid metabolism in hypertensive rats

2. Identification of Cd36 (Fat) as an insulin-resistance gene causing defective fatty acid and glucose metabolism in hypertensive rats

3. Membrane glycoprotein CD36: a review of its roles in adherence, signal transduction, and transfusion medicine

4. CD36 is a receptor for oxidized low density lipoprotein

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