Cytomegalovirus M45 Cell Death Suppression Requires Receptor-interacting Protein (RIP) Homotypic Interaction Motif (RHIM)-dependent Interaction with RIP1-Reference-Cited by-同舟云学术

Cytomegalovirus M45 Cell Death Suppression Requires Receptor-interacting Protein (RIP) Homotypic Interaction Motif (RHIM)-dependent Interaction with RIP1

Published:2008-06 Issue:25 Volume:283 Page:16966-16970
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Upton Jason W.,Kaiser William J.,Mocarski Edward S.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference38 articles.

1. The RIP kinases: crucial integrators of cellular stress

2. RIP1, a kinase on the crossroads of a cell's decision to live or die

3. Pathogen subversion of cell-intrinsic innate immunity

4. Apoptosis Induced by the Toll-Like Receptor Adaptor TRIF Is Dependent on Its Receptor Interacting Protein Homotypic Interaction Motif

5. RIP1 is an essential mediator of Toll-like receptor 3–induced NF-κB activation

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