Hepatic PCSK9 Expression Is Regulated by Nutritional Status via Insulin and Sterol Regulatory Element-binding Protein 1c-Reference-Cited by-同舟云学术

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Hepatic PCSK9 Expression Is Regulated by Nutritional Status via Insulin and Sterol Regulatory Element-binding Protein 1c

Published:2006-03 Issue:10 Volume:281 Page:6211-6218
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Costet Philippe,Cariou Bertrand,Lambert Gilles,Lalanne Florent,Lardeux Bernard,Jarnoux Anne-Laure,Grefhorst Aldo,Staels Bart,Krempf Michel

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference54 articles.

1. Monogenic hypercholesterolemia: new insights in pathogenesis and treatment

2. Proprotein and prohormone convertases: a family of subtilases generating diverse bioactive polypeptides1Published on the World Wide Web on 17 August 1999.1

3. Apolipoprotein B100 Metabolism in Autosomal-Dominant Hypercholesterolemia Related to Mutations inPCSK9

4. Common inactivating mutations in PCSK9 cause low LDL cholesterol

5. Low LDL cholesterol in individuals of African descent resulting from frequent nonsense mutations in PCSK9

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2. Proprotein convertase subtilisin/kexin type 9 deficiency in extrahepatic tissues: emerging considerations;Frontiers in Pharmacology;2024-07-30

3. Modification of lipoprotein metabolism and function driving atherogenesis in diabetes;Atherosclerosis;2024-07

4. Emerging clinical role of proprotein convertase subtilisin/kexin type 9 inhibition—Part one: Pleiotropic pro‐atherosclerotic effects of PCSK9;European Journal of Clinical Investigation;2024-06-26

5. The role of PCSK9 in heart failure and other cardiovascular diseases—mechanisms of action beyond its effect on LDL cholesterol;Heart Failure Reviews;2024-06-18

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