Extended Ischemia Prevents HIF1α Degradation at Reoxygenation by Impairing Prolyl-hydroxylation-Reference-Cited by-同舟云学术

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Extended Ischemia Prevents HIF1α Degradation at Reoxygenation by Impairing Prolyl-hydroxylation

Published:2010-06 Issue:24 Volume:285 Page:18217-18224
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Serra-Pérez Anna,Planas Anna M.,Núñez-O'Mara Analía,Berra Edurne,García-Villoria Judit,Ribes Antònia,Santalucía Tomàs

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference40 articles.

1. Induction of HIF–1α in response to hypoxia is instantaneous

2. Regulation of cancer cell metabolism by hypoxia-inducible factor 1

3. Hypoxia‐inducible factor‐1α (HIF‐1α) escapes O2‐driven proteasomal degradation irrespective of its subcellular localization: nucleus or cytoplasm

4. Targeting of HIF-α to the von Hippel-Lindau Ubiquitylation Complex by O 2 -Regulated Prolyl Hydroxylation

5. Mechanism of regulation of the hypoxia-inducible factor-1alpha by the von Hippel-Lindau tumor suppressor protein

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