APH-1 Interacts with Mature and Immature Forms of Presenilins and Nicastrin and May Play a Role in Maturation of Presenilin·Nicastrin Complexes-Reference-Cited by-同舟云学术

APH-1 Interacts with Mature and Immature Forms of Presenilins and Nicastrin and May Play a Role in Maturation of Presenilin·Nicastrin Complexes

Published:2003-02 Issue:9 Volume:278 Page:7374-7380
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Gu Yongjun,Chen Fusheng,Sanjo Nobuo,Kawarai Toshitaka,Hasegawa Hiroshi,Duthie Monica,Li Wenping,Ruan Xueying,Luthra Anchla,Mount Howard T.J.,Tandon Anurag,Fraser Paul E.,St George-Hyslop Peter

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference29 articles.

1. Cloning of a gene bearing missense mutations in early-onset familial Alzheimer's disease

2. Familial Alzheimer's disease in kindreds with missense mutations in a gene on chromosome 1 related to the Alzheimer's disease type 3 gene

3. Nicastrin modulates presenilin-mediated notch/glp-1 signal transduction and βAPP processing

4. Deficiency of presenilin-1 inhibits the normal cleavage of amyloid precursor protein

5. Facilitation of lin-12-mediated signalling by sel-12, a Caenorhabditis elegans S182 Alzheimer's disease gene

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