Abnormal expression of Tau in damaged oligodendrocytes of HLD1 mice
Author:
Affiliation:
1. Laboratory of Ion Channel Pathophysiology, Graduate School of Brain Science, Doshisha University, Kyotanabe-shi, Kyoto, Japan; Center for Research in Neurodegenerative Disease, Doshisha University, Kyotanabe-shi, Kyoto, Japan
Publisher
Medknow
Subject
Developmental Neuroscience
Reference12 articles.
1. Motor learning promotes remyelination via new and surviving oligodendrocytes;Bacmeister;Nat Neurosci,2020
2. Myelin debris regulates inflammatory responses in an experimental demyelination animal model and multiple sclerosis lesions;Clarner;Glia,2012
3. Overexpression of tau protein inhibits kinesin-dependent trafficking of vesicles, mitochondria, and endoplasmic reticulum: implications for Alzheimer's disease;Ebneth;J Cell Biol,1998
4. Glial response during cuprizone-induced de- and remyelination in the CNS: lessons learned;Gudi;Front Cell Neurosci,2014
5. Altered human oligodendrocyte heterogeneity in multiple sclerosis;Jäkel;Nature,2019
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