Oxidative Injury is a Common Consequence of BMPR2 Mutations-Reference-Cited by-同舟云学术

Oxidative Injury is a Common Consequence of BMPR2 Mutations

Published:2011-01 Issue:1 Volume:1 Page:72-83
ISSN:2045-8940
Container-title:Pulmonary Circulation
language:en
Short-container-title:Pulm. circ.

Author:

Lane Kirk L.¹,Talati Megha¹,Austin Eric²³,Hemnes Anna R.¹,Johnson Jennifer A.¹,Fessel Joshua P.¹,Blackwell Tom¹,Mernaugh Ray L.⁴,Robinson Linda¹,Fike Candice³,Roberts L. Jackson¹⁵,West James¹

Affiliation:

1. Department of MedicineVanderbilt University Medical CenterNashvilleTennesseeUSA

2. Department of GeneticsVanderbilt University Medical CenterNashvilleTennesseeUSA

3. Department of PediatricsVanderbilt University Medical CenterNashvilleTennesseeUSA

4. Department of BiochemistryVanderbilt University Medical CenterNashvilleTennesseeUSA

5. Department of PharmacologyVanderbilt University Medical CenterNashvilleTennesseeUSA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Pulmonary and Respiratory Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.4103/2045-8932.78107

Reference46 articles.

1. Heterozygous germline mutations in BMPR2, encoding a TGF‐beta receptor, cause familial primary pulmonary hypertension;Lane KB;Nat Genet,2000

2. Gross BMPR2 gene rearrangements constitute a new cause for primary pulmonary hypertension;Cogan JD;Genet Med,2005

3. Controllingcell fate by bone morphogenetic protein receptors;Dijke P;Mol Cell Endocrinol,2003

4. Direct signaling by the BMP type II receptor via the cytoskeletal regulator LIMK1;Foletta VC;J Cell Biol,2003

5. Mice expressing BMPR2R899X transgene in smooth muscle develop pulmonary vascular lesions;West J;Am J Physiol Lung Cell Mol Physiol,2008

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