Regional expression of the anesthetic-activated potassium channel TRESK in the rat nervous system
Author:
Publisher
Elsevier BV
Subject
General Neuroscience
Reference23 articles.
1. Current Protocols in Molecular Biology;Ausubel,1998
2. Pungent agents from Szechuan peppers excite sensory neurons by inhibiting two-pore potassium channels;Bautista;Nat. Neurosci.,2008
3. The ventilatory stimulant doxapram inhibits TASK tandem pore (K2P) potassium channel function but does not affect minimum alveolar anesthetic concentration;Cotten;Anesth. Anal.,2006
4. The two-pore-domain K+ channel, TRESK, is activated by the cytoplasmic calcium signal through calcineurin;Czirjak;J. Biol. Chem.,2004
5. TRESK two-pore-domain K+ channels constitute a significant component of background potassium currents in murine dorsal root ganglion neurones;Dobler;J. Physiol.,2007
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1. Critical contribution of the intracellular C-terminal region to TRESK channel activity is revealed by the epithelial Na+ current ratio method;Journal of Biological Chemistry;2023-06
2. The Special One: Architecture, Physiology and Pharmacology of the TRESK Channel;Cellular Physiology and Biochemistry;2022-11-25
3. Activation of TRESK background potassium channels by cloxyquin exerts protective effects against excitotoxic-induced brain injury and neuroinflammation in neonatal rats;Journal of Neuroimmunology;2022-07
4. Contribution of Neuronal and Glial Two-Pore-Domain Potassium Channels in Health and Neurological Disorders;Neural Plasticity;2021-08-12
5. TRESK channel contributes to depolarization-induced shunting inhibition and modulates epileptic seizures;Cell Reports;2021-07
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