Targeting mitochondria to oppose the progression of nonalcoholic fatty liver disease-Reference-Cited by-同舟云学术

Targeting mitochondria to oppose the progression of nonalcoholic fatty liver disease

Published:2019-02 Issue: Volume:160 Page:34-45
ISSN:0006-2952
Container-title:Biochemical Pharmacology
language:en
Short-container-title:Biochemical Pharmacology

Author:

Grattagliano Ignazio^ORCID,Montezinho Liliana P.,Oliveira Paulo J.^ORCID,Frühbeck Gema,Gómez-Ambrosi Javier,Montecucco Fabrizio,Carbone Federico^ORCID,Wieckowski Mariusz R.,Wang David Q.-H.,Portincasa Piero

Funder

Marie Skłodowska-Curie

Publisher

Elsevier BV

Subject

Pharmacology,Biochemistry

Reference163 articles.

1. Is it time to change NAFLD and NASH nomenclature?;Bellentani;Lancet,2017

2. Asia-Pacific Working Party on Non-alcoholic Fatty Liver Disease guidelines 2017-Part 1: definition, risk factors and assessment;Wong;J. Gastroenterol. Hepatol.,2018

3. Hypertension, diabetes, atherosclerosis and NASH: cause or consequence?;Lonardo;J. Hepatol.,2018

4. PNPLA3-associated steatohepatitis: toward a gene-based classification of fatty liver disease;Krawczyk;Semin. Liver Dis.,2013

5. Global burden of NAFLD and NASH: trends, predictions, risk factors and prevention;Younossi;Nat. Rev. Gastroenterol. Hepatol.,2018

Cited by 51 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Peridroplet mitochondria are associated with the severity of MASLD and the prevention of MASLD by diethyldithiocarbamate;Journal of Lipid Research;2024-08

2. The relationships between high-fat diet and metabolic syndrome: Potential mechanisms;Food Bioscience;2024-06

3. PBAT-modified starch blended film extract induces in vitro toxicity in L-02 cells: induction of oxidative stress, inflammation, and modulation of AMPK pathway;Drug and Chemical Toxicology;2024-05-10

4. Proteomic study of gamma-oryzanol preventive effect on a diet-induced non-alcoholic fatty liver disease model;The Journal of Nutritional Biochemistry;2024-05

5. Contributing roles of mitochondrial dysfunction and hepatocyte apoptosis in liver diseases through oxidative stress, post-translational modifications, inflammation, and intestinal barrier dysfunction;Cellular and Molecular Life Sciences;2024-01-12