Renal heme metabolism in hereditary tyrosinemia: use of succinylacetone in rat renal tubules
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Biochemistry,Biophysics
Reference26 articles.
1. Deficiency of fumarylacetoacetase in a patient with hereditary tyrosinemia
2. Enzyme Defect in a Case of Tyrosinemia Type I, Acute Form
3. Effects of succinylacetone on methyl α-d-glucoside uptake by the rat renal tubule
4. Hereditary Tyrosinemia and the Heme Biosynthetic Pathway. PROFOUND INHIBITION OF δ-AMINOLEVULINIC ACID DEHYDRATASE ACTIVITY BY SUCCINYLACETONE
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2. Immunizations for Patients With Metabolic Disorders;Pediatrics;2006-08-01
3. Determination of renal porphyrin handling in rats suffering from different kinds of chronic renal failure (CRF): Uranyl nitrate (UN) induced fibrosis or 5/6-nephrectomy (5/6NX);Experimental and Toxicologic Pathology;2003-01
4. Tissue distribution of succinylacetone in the rat in vivo: a possible basis for neurotoxicity in hereditary infantile tyrosinemia;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;1993-10-20
5. Physiological basis for an animal model of the renal Fanconi syndrome: use of succinylacetone in the rat;Clinical Science;1992-07-01
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